Navicular Syndrome
By Carla Huston, BES
Navicular syndrome is so called because a number of conditions may cause
the pathological changes and clinical signs, and many structures may be
involved. The navicular bone lies on the back of the coffin bone, with
the navicular bursa, a small fluid-filled sac, between them. The small
boat-shaped bone serves as a pulley for the deep flexor tendon that
glides along its underside. Navicular syndrome is a degenerative
process that can affect the bone, bursa or tendon.
This condition occurs almost exclusively in the front feet, and usually
affects both feet. The first signs of navicular are a shortening of the
stride and slight unevenness on turns. Because both forefeet are
affected simultaneously, obvious lameness is not commonly observed.
Instead, the horse appears reluctant to go forward properly or lengthen
his stride. Pointing is another common sign; the horse will stand at
rest with one leg extended, the weight resting on the toe. When the
foot is pressure tested, the horse will usually indicate pain at the
heels. X-rays do not always detect early signs of navicular since the
soft tissues are usually the first problem areas. The cartilage on the
underside of the navicular bone and the deep flexor tendon become
inflamed first.
The causes of navicular, unfortunately, are usually manufactured. First,
horses with small feet (too small for their weight) are predisposed to
this condition. The support structures in the hoof are designed by
nature to absorb shock and carry the horse; the smaller the hoof in
relation to the horse's size, the greater the shock to the navicular
bone. Breeders often select for small feet, believing them more
"attractive." A second cause is using the horse in stressful athletic
activities for which they are not suited. Circulation trouble is the
first step in the breakdown. The reduced blood flow weakens the
structure and leads to the deterioration. Horses that undergo a fair
amount of percussion on the forefeet are inclined to experience
navicular syndrome.
No single treatment is uniformly successful in treating navicular. These
therapies treat the symptoms, but the syndrome is irreversible. The
possibilities range from drugs to corrective farriery work and surgery
to retirement. Bute and other anti-inflammatory drugs will alleviate
pain. The administration of warfarin in the feed may help some horses.
This drug prolongs the blood clotting time, but is potentially dangerous,
and permanent medication is usually necessary. Why the drug works is
not known. Also occasionally successful is the administration of
isoxuprine, also delivered through the feed or in a paste. The
peripheral blood vessels are dilated, improving circulation. Isoxuprine
is usually only successful in the early stages of the syndrome. The foot
must be properly trimmed, with the toes kept short and the foot balanced.
Therapeutic shoeing may include an egg bar shoe with wedge pads, or the
newly developed Tennessee Navicular shoe. Neurectomy is a surgical
option for the navicular horse. This procedure involves removing a
small piece of the two nerves that innervate the area. The nerves may
regenerate, however, and daily care of the foot must be observed to
recognize any injuries to the foot. Finally, for the advanced navicular
syndrome, retirement may be the only recourse. This should include daily
turnout to maintain regular bloodflow and promote healthy hoof growth.